ABSTRACT
The COVID-19 pandemic is the most disastrous health event of the twenty-first century with significant impacts on the economy, institutions, organizations and people's well-being and lifestyle. Risk perception is a knowledge process that manifests itself in risk behavior and reflects people's experiences, beliefs and social influences. The management of risk perception is essential in contributing to people's protective behaviors in the presence of health risks. In Cuba, one of its significant effects is reflected in the psychology of individuals, despite policies to mitigate its consequences. Minimizing COVID-19 transmission requires the application of effective policies that contribute with adequate perceptions, depending on the risk factors. The goal of this research is to develop a system of actions that can contribute to the transition of a favorable scenario for the management of risk perception regarding COVID-19 in Cuba according to the new normality and through the use of a scenario planning methodology. The findings allow characterizing the current scenario and proposing actions and indicators in order to achieve the most favorable scenario for risk perception management related to COVID-19 from a geographic perspective. © The Author(s), under exclusive license to Springer Nature Switzerland AG 2022.
ABSTRACT
Introduction: Coronavirus disease 2019 (COVID-19), caused by a new coronavirus. Severe acute respiratory syndrome coronavirus 2 is having a devastating impact throughout the world. Its pathophysiology is the result of direct cytopathic effects of SARS-CoV-2 on respiratory epithelium, endothelium, other organ-specific cell types and the subsequent induction of a pro-inflammatory cytokine storm and dysregulated adaptive immunity, causing severe tissue damage. Objective(s): To describe autoimmune characteristics present during SARS-Cov-2 infection. Method(s): A literature review on COVID-19 and autoimmune manifestations was carried out. The articles were selected from a search of the following websites: Pubmed, Medline, SciELO. An analysis and summary of the reviewed information was made. Analysis and synthesis of information: Viruses have been considered the main infectious agents that trigger autoimmune phenomena in genetically susceptible individuals. SARS-CoV-2 has already been linked to a long list of autoinflammatory and autoimmune conditions that can be induced by various mechanisms proposed to explain virus-induced autoimmunity. In this pathology, the most described, is molecular mimicry. COVID-19 shares similarities with autoimmune diseases in its clinical manifestations, pathophysiological mechanisms and therapeutic response, such powerful immune reactions participate in the pathogenesis of both entities. Conclusion(s): SARS-CoV-2 infection may represent the complex and interrelated link between infections and autoimmunity.Copyright © 2022, Editorial Ciencias Medicas. All rights reserved.
ABSTRACT
Introduction: Coronavirus disease 2019 (COVID-19), caused by a new coronavirus. Severe acute respiratory syndrome coronavirus 2 is having a devastating impact throughout the world. Its pathophysiology is the result of direct cytopathic effects of SARS-CoV-2 on respiratory epithelium, endothelium, other organ-specific cell types and the subsequent induction of a pro-inflammatory cytokine storm and dysregulated adaptive immunity, causing severe tissue damage. Objective(s): To describe autoimmune characteristics present during SARS-Cov-2 infection. Method(s): A literature review on COVID-19 and autoimmune manifestations was carried out. The articles were selected from a search of the following websites: Pubmed, Medline, SciELO. An analysis and summary of the reviewed information was made. Analysis and synthesis of information: Viruses have been considered the main infectious agents that trigger autoimmune phenomena in genetically susceptible individuals. SARS-CoV-2 has already been linked to a long list of autoinflammatory and autoimmune conditions that can be induced by various mechanisms proposed to explain virus-induced autoimmunity. In this pathology, the most described, is molecular mimicry. COVID-19 shares similarities with autoimmune diseases in its clinical manifestations, pathophysiological mechanisms and therapeutic response, such powerful immune reactions participate in the pathogenesis of both entities. Conclusion(s): SARS-CoV-2 infection may represent the complex and interrelated link between infections and autoimmunity. Copyright © 2022, Editorial Ciencias Medicas. All rights reserved.
ABSTRACT
The COVID-19 pandemic resulted in a worldwide sports shutdown, leading to a reduced athletes' workload, which is likely to increase the risk of injury after return to play when coupled with the expected increase in competitive density after the confinement period. in this regard, the knee is one of the joints with the highest injury prevalence among female soccer players, being the injury of the anterior cruciate ligament (ACL) one of the most frequent and severe. Thus, the present study aimed to develop an ACL injury prevention program that female soccer players could carry out during confinement periods. After reviewing the scientific literature, it was observed that strength training (with a main focus on the posterior chain), improved landing technique, lumbopelvic stability, and flexibility are associated with a lower risk of ACL injury in women. The program focuses on modifiable risk factors, so it should serve as a guide that can be adapted according to each context: individual characteristics, initial conditions, and evolution of the subject during development. Therefore, this study presents an intervention aiming at reducing the probability of ACL injury in female soccer players, after confinement periods, by directly influencing mechanisms of ACL injury previously reported for female soccer players.
ABSTRACT
An innovative educational management proposal is presented to address the effects of COVID-19 and to guide actions towards a human organizational culture at CIDE. © 2020 Universidad Nacional. All rights reserved.